<p> </p><p>

Chapter ONE

</p><p>1.0 &nbsp; &nbsp; Introduction</p><p>1.1 &nbsp; &nbsp; Atherosclerosis</p><p>1.2 &nbsp; &nbsp; Pathogenesis</p><p>1.2.1 Atherogenesis</p><p>1.2.1.1 Initiation</p><p>1.2.1.2 Progression</p><p>

Chapter TWO

</p><p>2.0 &nbsp; &nbsp; Biochemical basis</p><p>2.1 &nbsp; &nbsp; Types of &nbsp;plaque</p><p>2.1.1 Features plaques</p><p>2.1.1.1 Lipid-rich core</p><p>2.1.1.2 Cell death</p><p>2.1.1.3 Calcification</p><p>2.1.1.4 &nbsp; Neovascularization and intraplaque hemorrhage</p><p>2.1.1.5 &nbsp; Vascular remodeling and luminal stenosis</p><p>2.1.1.6 Plaque rupture</p><p>2.1.1.7 Nonfatal thrombosis</p><p>2.1.1.8 &nbsp; Fatal thrombosis</p><p>2.1.1.9 Fibrin and platelets</p><p>2.1.1.10 Contribution of bone marrow-derived cells</p><p>2.1.2 Immunohistochemistry in plaque</p><p>

Chapter THREE

</p><p>3.0 &nbsp; &nbsp; Inflammation in</p><p>3.1 &nbsp; &nbsp; Inflammation and the initiation</p><p>3.2 &nbsp; &nbsp; Inflammation causes various forms of plaque disruption</p><p>3.3 &nbsp; &nbsp; Inflammation as a therapeutic target in</p><p>3.4 &nbsp; &nbsp; Inflammatory markers as gauges risk</p><p>3.5 &nbsp; &nbsp; Itherosclerosis and cardiovascular disease</p><p>

Chapter FOUR

</p><p>4.0 &nbsp; &nbsp; Physical activity in prevention</p><p>

Chapter FIVE

</p><p>5.0 &nbsp; &nbsp; Conclusion and recommendation</p><p>5.1 &nbsp; &nbsp; Conclusion</p><p>5.2 &nbsp; &nbsp; Recommendation</p> <br><p></p>